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吴旭平*

吴旭平博士,南京市第二医院(江苏省传染病医院)。2011年获瑞典乌普萨拉大学肿瘤学博士学位,2011-2012于瑞典乌普萨拉大学从事博士后研究。先后入选南京市中青年拔尖人才培养对象(卫生拔尖人才)、江苏省双创博士、江苏省“333高层次人才培养工程”培养对象、江苏省“六大人才高峰计划”、江苏省十三五科教强卫工程等多项人才项目;并先后主持国家自然科学基金、南京市医学科技发展项目杰出青年基金等项目。在肝炎、Gilbert综合征和肿瘤等疾病的检测工作研发方面取得了一系列成果,多项工作在ACS Appl. Mater. Interfaces, Biosensors and Bioelectronics, Br JCancer, International Journal of Molecular MedicineMolecular Cancer TherapeuticsInternational Journal of Oncology等国际期刊上发表,以第一申请人牵头的相关成果多项。

 

 

 

研究方向:肿瘤及分子检测

联系方式:南京市第二医院,钟阜路1-1号,电话:02583626181

 

主要在研项目:江苏省卫计委十三五科教强卫工程青年人才项目,QNRC2016055, 用于诊断疑难性Gilbert综合症基因芯片技术的研发

 

 

代表性论文:

代表性论文:

[1] Song J, Sun M, Li J, Zhou D, and Wu X*, “Three-dimensional polyacrylamide gel-based DNA microarray method effectively identifies UDP-glucuronosyltransferase 1A1 gene polymorphisms for the correct diagnosis of Gilbert's syndrome,” International Journal of Molecular Medicine37, 575-80(2016).

[2]Feng J, Kang W, Wu X, Wang S, and Liu F*,“Quantitative Detection and Real-Time Monitoring on Endogenous mRNA in Single Live Cell Level Using Ratiometric Molecular BeaconACS Appl. Mater. Interfaces (2019), 11, 32, 28752-28761.

[3] Shan Y, Wang B, Huang H, Jian D, Wu X, Xue L, Wang S*, Liu F*, “On-site quantitative Hg2+measurements based on selective and sensitive fluorescence biosensor and miniaturized smartphone fluorescence microscope” Biosensors and Bioelectronics(2019), 162: 238-247

[4] Wu X*, Sooman L, Wickström M, Fryknäs M, Dyrager C, Lennartsson J, and Gullbo J, “Alternative cytotoxic effects of the postulated IGF-IR inhibitor picropodophyllin in vitro,” Molecular Cancer Therapeutics12, 1526-36(2013).

[5] Wu X*, Sooman L, Lennartsson J, Bergström S, Bergqvist M, Gullbo J, and Ekman S, “Microtubule inhibition causes epidermal growth factor receptor inactivation in oesophageal cancer cells,” International Journal of Oncology42, 297-304(2013).

[6] Wu X*, Smavadati S, Nordfjäll K, Karlsson K, Qvarnström F, Simonsson M, Bergqvist M, Gryaznov S, Ekman S, and Paulsson-Karlsson Y, “Telomerase antagonist imetelstat inhibits esophageal cancer cell growth and increases radiation-induced DNA breaks,” Biochimica et BiophysicaActa – molecular cell research1823, 130-2135(2012). 

[7] Wu X*, Wanders A, Wardega P, Tinge B, Gedda L, Bergstrom S, Sooman L, Gullbo J, Bergqvist M, Hesselius P, Lennartsson J, and Ekman S, “Hsp90 is expressed and represents a therapeutic target in human oesophageal cancer using the inhibitor 17-allylamino-17-demethoxygeldanamycin,” Br J Cancer100, 334-43(2009).